patellofemoral syndrome is characterized by a algia at retrorotulea more often localized on the lateral side of the femoral trochlea.
The rumination of this article, takes as its premise the idea that the disbanding of the patella (if not characterized by a significant lack of restraint anatomic lateral side of the pulley-dysplasia-) not due to a lack of 'activation of the vastus medialis but, paradoxically, by insufficient component coattazione directed posteriorly.
This minor component could result from a micro-slip front of the tibia during the mechanism of flexion or bending due to the inadequacy of the flexor muscles of the thigh. The reduction of this component could be the cause back by the increased tendency of going to support the patella more laterally creating a conflict patellofemoral joint in the side.
E 'undeniable noted that this reasoning is exactly opposite to what is traditionally understood: the sidrome patellofemoral joint is not seen as an excess of compression, but as a lack of compression. More specifically I would say a lack of organized compression because it is clear that compression is not organized inevitably leads to a degradation of tissues.
If we imagine the back faces of the patella with a geometry complementary to the femoral trochlea, we can better understand that, when the compression force is narrowing, the patella loses its stability, its intimacy with the femur allows you to "float" and then to veer where the component of the quadriceps decides to take .
Increase this component through the quadriceps, even when it was possible to block the reinforcement of the vastus medialis, would inevitably lead to a simultaneous increase in the heeling side component.
Hence the idea of \u200b\u200bworking on the action of the hamstrings are going to contain a possible uncontrolled movement of the sliding front of the tibia, a movement that we know are of great magnitude during the movement of knee extension.
